Thermoregulatory responses to combined moderate heat stress and hypoxia
Low DA, Bailey TG, Timothy Cable N, Jones H
Microcirculation (New York, N.Y. : 1994) · 7 citas
Cómo se estudió
- Diseño
- Ensayo clínico controlado (clasificado por nuestro filtro de IA)
- Estudiado en
- Personas
- Resultado principal
- Marcadores de salud y funcionamiento
Quién lo pagó
- Financiación
- Financiación independiente
- Gobierno
- Seventh Framework Programme
- Subvenciones
- Seventh Framework Programme (239143)
Según 1 financiador declarado.
Publicación
- Publicado
- 2016-07-15 · Microcirculation · vol. 23 · número 7 · págs. 487–494
- Editorial
- Wiley
- Citado
- 7 citas · más que el 69% de estudios similares · 0.6× el promedio de su campo
- Referencias
- 40 trabajos
- Acceso
- Lectura gratuita
- Áreas de investigación
- Thermoregulation and physiological responses · Thermal Regulation in Medicine · Infrared Thermography in Medicine
- Palabras clave
- Medicine, Hypoxia (environmental), Forearm, Internal medicine, Thermoregulation, Sudomotor, Heart rate, Cardiology, Skin temperature, SWEAT, Core temperature, Endocrinology, Anesthesia, Blood pressure, Oxygen, Surgery, Chemistry, Biomedical engineering
- MeSH
- forearm, thorax, skin, humans, body temperature regulation, sweating, skin temperature, heat-shock response, regional blood flow, adult, male, young adult, hypoxia
4 autores
- David A. Low🇬🇧 · autor de correspondenciaLiverpool John Moores University
- Tom G. Bailey🇬🇧Liverpool John Moores University
- Nigel Timothy Cable🇶🇦🇬🇧Aspire Academy; Liverpool John Moores University
- Helen E Jones🇬🇧Liverpool John Moores University
Resumen
El texto original del estudio, en el idioma en que se publicó.
Objective
The aim of this study was to examine the cutaneous vascular and sudomotor responses to combined moderate passive heat stress and normobaric hypoxia.
Method
Thirteen healthy young males, dressed in a water-perfused suit, underwent passive heating (Δcore temperature ~0.7°C) twice (Normoxia; 20.9% O2 and Hypoxia; 13% O2 ). Chest and forearm skin blood flow (SkBF; laser-Doppler flux) and sweat rate (SR; capacitance hygrometry), core (intestinal pill), and skin temperatures, were recorded.
Results
Hypoxia reduced baseline oxygen saturation (98±1 vs 89±6%, P-1 , P<.01). During heating, mean body temperature (T¯BODY) thresholds for SkBF (P=.41) and SR (P=.28) elevations were not different between trials. The SkBF: T¯BODY linear sensitivity during the initial phase of heating was lower at the chest (P=.035) but not different at the forearm (P=.17) during hypoxia. With increasing levels of heating chest SkBF was not different (P=.55) but forearm SkBF was lower (P<.01) during hypoxia. Chest (P=.85) and forearm (P=.79) SR: T¯BODY linear sensitivities were not different between trials.
Conclusion
While sudomotor responses and the initiation of cutaneous blood flow elevations are unaffected, hypoxia differentially effects regional SkBF responses during moderate passive heating.
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